Volume 3, Issue 1 (3-2014)                   Plant Pathol. Sci. 2014, 3(1): 75-82 | Back to browse issues page

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ALISHIRI A, RAKHSHANDEHROO F. (2014). The Role of Salicylic Acid in Plant Resistance Against Plant Pathogens . Plant Pathol. Sci.. 3(1), 75-82.
URL: http://yujs.yu.ac.ir/pps/article-1-61-en.html
Ph.D. Student and of Plant Pathology, Department of Plant Pathology, Science and Research Branch, Islamic Azad University, Tehran, Iran , alishiria@yahoo.com
Abstract:   (21246 Views)
Salicylic acid is a kind of phenolic acids and a derivate of salicylate hormone that produced in plants in response to the large numbers of plant pathogens and is necessary for inducing systemic acquired resistance. Vast numbers of studies have done by researchers on the salicylic acid function in inducing plant resistance genes specially those that are responsible for pathogenesis related protein encoding. Results indicated to the effective role of this hormone in inducing innate resistance against pathogenic agents in plants. Changes that block the salicylic acid production in plants increase the disease susceptibility to plant pathogens. Transcription factors belonging to the WRKY, TGA and MYB families are involve in salicylic acid dependent resistance to plant pathogenic agents. Salicylic acid induces the pathogenesis related proteins through the NPR1 dependent pathway during hypersensitive response and systemic acquired resistance reactions. Signaling pathway, independent from NPR1 is a different form of salicylic acid dependent signal transduction and is require for specific resistance against pathogens. Salicylic can also induce the RNA-Dependent-RNA Polymerase gene expressions and play an important role in RNA silencing. Totally, with having knowledge about the salicylic acid biology and its signal transduction pathways in plants it is possible to increase the crop yields with increasing the resistances potential to stresses.
Full-Text [PDF 114 kb]   (2366 Downloads)    
Type of Study: Extentional | Subject: Special
Received: 2013/10/7 | Accepted: 2014/03/18

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